We can work on Virtual Currency

Quick—how much cash do you have on you right now? Check your wallet, purse, and pockets, and see what you have. Do you have enough to make change for a twenty-dollar bill? Not so long ago, you could ask a roomful of people that question, and most folks would have the cash on hand to make change. These days, it’s less common. Think about the last five or six purchases you made. Did you pay with cash? Chances are good that you used a debit card or a credit card instead. Consider the possibility that someday debit and credit cards will disappear, too.

Technology has had a profound impact on our view of the necessity of cash. As we move towards a “cashless” society, the banking system is also challenged to adapt its processes and the services it provides.

Go beyond what you learned in the course module, and research some of the forms of “virtual currency” or cash alternatives that are currently available to consumers. Some of these include digital currencies, EFT, ACH transactions, iPay, Google Wallet, ePayments, direct draft.

Discuss
Briefly explain the alternative form of payment that you researched and how it works as an alternative to traditional cash.
What are the advantages of this form of payment?
Are there disadvantages or risks associated with the form of payment that you researched?
As a consumer yourself, do you use any forms of cash alternatives? Which ones? Do you have concerns about the security of this payment method, and if so, what can you do to mitigate these?

Sample Solution

2nd and 3rd toe. Autonomic neuropathy occurs when blood is shunted away from peripheral cutaneous capillary beds, which may occur in patients with PAD associated with diabetes. Motor neuropathy leads to changes in gait and thus more pressure on one leg, leading to ulceration. The loss of protective sensation and proprioception resulting in increased force with each step may lead to formation of calluses at pressure areas, which decreases elasticity and increases skin ischemia. Patient has diabetic neuropathy. Acute occlusion of a lower extremity artery may occur with chronic PAD and development of an acute thrombosis. It is called “critical limb ischemia” when the chronic development of peripheral artery occlusive disease in the lower extremity becomes severe. It is manifested by ischemic ulcers of the foot. Ischemic ulcers often begin as minor traumatic wounds and fail to hail because the blood supply is insufficient to meet the increased demands of the healing tissue. Ulcerations caused by ischemia are typically lcated at the termination of arterial branches. They are commonly found on the tips of the toes and between the digits. They can also form at increased focal pressure, such as lateral malleolus and metatarsal heads. In addition to ulcers, patient can present a gangrenous digit or foot. Gangrene can either be dry or wet. Dry gangrene is characterized by a hard, dry texture, often with a clear demarcation between viable and black, necrotic tissue. This form of gangrene is common in patients with PAD. Wet gangrene is characterized by its moist appearance, gross swelling, and blistering. Wet gangrene is a surgical emergency. Pt presented in the ED with wet gangrene, and dry gangrene post op. (Neschis, 2016) Ischemia sufficient to threaten a limb occurs when arterial blood flow is insufficient to meet the metabolic demands of resting muscle or tissue. Once patient’s body is unable to maintain the metabolic needs, it goes into anaerobic metabolism, which is seen in labs as high lactic acid. Acute thrombosis of sites of stenosis in which the blood flow impairment was hemodynamically significant can occur and present with acute symptoms., such as pain, pallor, paresthesias, paralysis, pulselessness, and poikilothermia(coldness). Symptoms of parethesias and paralysis may indicate advanced ischemia that is affecting nerve pathways of the extremity. Patient had ischemic ulcer in L foot, and signs of parethesias, pulselessness, and pain. (Baird, 2016 p590, Porth, 2011 p415) The presence of ischemia from occlusion to a lower extremity influences the timing of revascularization, debridement, and definitive coverage/closure. Wounds will not be able to heal as well as a result of ischemia and may lead to necrotizing of the soft tissue. It may come in the form of cellulitis, myositis, and fasciitis. Necrotizing cellulitis include anaerobic infection and Meleny’s syngergistic gangrene. Anaerobic cellulitis can be divided into clostridial anaerobic cellulitis and non-clostridial anaerobic cellulitis. Clostridial anaerobic cellulitis is usually caused by C. perfringens. These organisms may be introduced into the subcutaneous tissue via trauma, surgical contamination, or spread of infection fro>

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